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Deaths and cancer incidence (solid cancers and all cancers) were extracted from LSS [ 25, 26 ]. Excess ERR was calculated by subtracting 1.2 for Hiroshima and Nagasaki from that of the unexposed rate in the same areas. For deaths from selected solid cancers (Table 2), the average excess relative risk for post-bomb ex-Gensakusha (caught in the rain) is of the order of 8. Numbers for cancers are smaller and are less (by an order of magnitude) than numbers for deaths. If LNT is true, for every 10,000 people who were exposed, about 8-8.5 cancer deaths would have been expected. But the expected number of cancer deaths is 18.3 (0.1% of the area exposed, or 18.1 for the 11 districts in Hiroshima and Nagasaki), instead of the actual number of cancer deaths (18.3), which means that excess cancer deaths were actually 8 to 9 times higher than expected.
Babies in Hiroshima and Nagasaki had a higher risk of death from cancer (age 35-39) than those born before or after: 4.9% vs 3.5% [ 27 ]. The risk of death from all causes is also higher for the late-born babies. A weighted risk from cancer is 17.1% vs 11.3%. My re-analysis of the LSS data for all ages shows a similar pattern (Table 3). In other words, being born after the atmospheric atomic bomb was nearly as dangerous for cancer mortality as being born after the atomic bomb.
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nuke ip death v6.6.6 downloadPatients with ankylosing spondylitis who take antirheumatic drugs after beginning tumor necrosis factor-α inhibitors have a lower rate of infection-related hospitalization compared with patients who take these drugs before tumor necrosis factor-α inhibitor initiation.
Tumor necrosis factor-α inhibitors (TNFi) have been shown to be effective in the treatment of ankylosing spondylitis (AS). However, there is a need to understand long-term outcomes in AS patients taking TNFi who have started treatment after 1996. A total of 1812 AS patients who had started TNFi therapy were identified from a large database. Information was obtained from the database on demographics, AS disease characteristics, concomitant medication use, comorbid conditions, TNFi use, and hospitalizations for infection. The mean duration of followup was 5.5 years, and 595 (33%) of the 1812 patients had begun TNFi therapy after 1996. Patients who started TNFi after 1996 were younger, had a shorter time since symptom onset, and were less likely to have peripheral arthritis. Unadjusted analyses suggested that patients who took TNFi after 1996 were less likely to be hospitalized for an infection than were those who took TNFi before 1996. The likelihood of being hospitalized for infection decreased with
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